The Morgellons Experience; Hopeless, or is it?

Morgellons Sufferers Speak Out

In the nearly 6 years since I have been helping Morgellons sufferers I have heard thousands of stories about how day-to-day life is for the Morgellons sufferer.  Here, we offer a typical story, written by the victim, describing their horrific experiences with doctors, dermatologists and general healthcare practitioners.

If you have Morgellons disease, you do not need to suffer; there is a way out.

_______________________________________________________

Hi all,

I am a 23 year old female originally from Russia; currently living
in WI, USA. Ironically, my interest in Morgellons started way before I
caught it myself (I just read a couple articles on the weird fiber
disease and was trying to figure out if it was for real). But then, two
year ago, back in April-May of 2010 I got a weird sore on my neck. As a
teenager I’ve had pretty bad and slow-healing acne, so I figured out
this was just a nasty zit. The sore, however, continued to grow and
ulcerate, and so I went to a doctor. The urgent care intern told me
excitedly that she knows exactly what it is- MRSA! and prescribed me
sulfa antibiotics. They never helped, and more sores started popping up
on my neck. They all started the same- first something looking like a
pimple but with a hard center (impossible to squeeze out); a day or two
later, the center of the “pimple” darkened and “rotted away”, making
this ugly black/brown scab. At this point the scab was nearly
impossible to tear off, but a few days later it would soften up on its
own and fall off, forming an ulcer. The ulcer would then either never
heal or heal very slowly, while constantly weeping and oozing out stuff.
I also noticed some weird white granules that came out of the ulcers,
but I thought that those perhaps were clusters of my own body cells, so I
paid no big attention to them. The lesions became worse and worse
though, and my next dermatologist, after a quick check-up, rudely kicked
me out and put “excoriation” as diagnosis.
Thus became my long battle with dermatologists. The next one made
me take at least 2 courses of antibiotics before she would ever consider
to try something else (i.e. anti-fungals). The biopsies so far showed
no infection except some staph, etc. By that point I was searching
through hundreds and hundreds of dermatological articles on various skin
diseases, and obsessively comparing pictures of those to mine.
Finally, I decided that I’ve got sporotrichosis. This was a pretty
rational conclusion, especially taking into account that I worked in a
lab during spring break where I was exposed to hay, moss, etc, which was
also genetically altered. (Sporotrichosis is a fungal infection caused
by a tiny organism that lives on certain types of vegetation.) At
first I tried potassium iodide (SSKI) and it seemed to help somewhat
(the lesions turned from HORRENDOUS to pretty bad). And then, after a
lot of bureaucratic procedures I finally got my itraconazole and started
taking it as directed, 100 mg/day. Pretty soon my sores started
healing and clearing up, and new ones did not appear. I was really happy
and intended to finish the whole itraconazole course (3 months or so),
but what happened instead was a big surprise! I got pregnant! Alas, of
course, I could not take the anti-fungal anymore, and the lesions
slowly started creeping up back. I was afraid that both me and the baby
would be eaten by them months before I give birth, but for some reason
they did not really worsen during the whole pregnancy- i.e. the old ones
kept healing but then new small ones would pop up in a different spot.

Is Morgellons contagious?

So I continued researching the internet for other possible
diagnoses. Plus, I work in a lab and have an access to a really good 3D
optical microscope, so I checked a couple slides with the lesion skin
samples, and there were black threads embedded in the skin. That, of
course, linked directly to Morgellons, but after reading about “Intense,
pruitic itching” I dismissed that disease as a manifestation of my
paranoia, since I did not experience any itching whatsoever.

After giving birth though, I did not even consider breastfeeding-
the sores started coming back at an alarming rate, and I needed to get
rid Morgellons baby's handof them asap! Again, after a lot of tedious insurance procedures I
got an itraconazole refill. This time, however, the lesions disappeared
rather reluctantly and some of them kept coming back. I started to
suspect that there was something more to it than a “simple”
sporotrichosis (actually a pretty complicated disease, with the
yeast-shaped fungus cells often being missed in biopsies even with
special stains), so I thought that with my luck I got a case of mutated
sporotrichosis that is drug-resistant. However, I continued to take
samples of my skin and scabs to the lab and study them in detail under
the microscope. What I observed was shockingly bizzare- white, blue,
and thicker black threads all around my skin and mostly in/on the sores;
the scabs have gel-like “roots”; there are threads embedded in my
FINGERNAILS and there are also those weird creatures (the granules that
could be squeezed out) that all have a little piece of fiber sticking
out (follicles? antennas?) Finally, I googled up some close-up
Morgellons sores and scabs. Did something strike you as WEIRD about
them when you first got to see them in a bright light? They all have
these circular and curvy grooves on them that form patterns eerily
similar to something like an elm seed (see pic). The center is either a
blood bubble or a dry black dot that is impossible to pick off (but if
you do manage to, try picking it up with a magnetized needle- those
“dots” seem to be some kind of iron collections (from blood?) and might
serve as a means of “communication” between individual lesions. That
happened to me on several occasions- one sore starts itching or hurting,
and then suddenly stops, and another one on the same hand but a
distance away would do the same thing.

In conclusion, I would like to thank the people who created this
forum, since I really need some support (instead of being told that I am
crazy and sent away). I sincerely hope that (somewhat graphic) details
of my story would help somebody to figure out more about their own
conditions or perhaps, even, put all clues together to come at some
definitive conclusion about what they are. In meantime, I am wishing
everyone struggling with this nasty evil thing to find a method that
works for them, and to have enough strength and self-esteem to cope with
the frightening reality.

Guaranteed Morgellons Disease Treatment

You are here to find a solution to your Morgellons infection.  You have found it. NutraSilver simply works and we guarantee that for 60 days. Do not suffer another day. There is no risk to you because your results are guaranteed. Morgellons done. Here is Connie’s story and how she conquered Morgellons disease using NutraSilver.

  • NutraSilver is a fast and effective Morgellons Disease Treatment
  • Safe and Guaranteed to Work on your Morgellons Disease Symptoms, money-back guarantee
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  • Watch your Lesions Heal Quickly and fall off naturally
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Morgellons Proven to be a Real Disease

Millions of People Infected!

Morgellons Disease is characterized by bizarre symptomology such as experiencing biting sensations on the skin, painful skin eruptions, strange fibers growing from the skin, unexplained body pain and neurological deficits (1,2). To make matters worse, patients are often bit by spiders, ticks, fleas, mites and other insects while suffering from the illness.

It is estimated that millions of people worldwide suffer from this unexplained affliction with medical personal quickly to dismiss their symptoms as ‘delusional parasitosis’ or ‘delusional infestation’. Medical authorities often time blame the Morgellons sufferer for inflicting wounds on themselves and believe the fibers simply emanate from clothing.

After thousands of complaints to the Center for Disease Control and Prevention (CDC), In January of 2008, the CDC paid researchers over $300,000 to conducted a 3-year study in to Morgellons. The researchers erroneously concluded that “No common underlying medical condition or infectious source was identified, similar to more commonly recognized conditions such as delusional infestation”(3) and hence indirectly concluded that Morgellons was not a disease and was in fact a psychological disorder. The CDC chimed in as well stating, “This comprehensive study of an unexplained apparent dermopathy demonstrated no infectious cause and no evidence of an environmental link. There was no indication that it would be helpful to perform additional testing for infectious diseases as a potential cause. Future efforts should focus on helping patients reduce their symptoms through careful attention to treatment of co-existing medical, including psychiatric conditions, that might be contributing to their symptoms”(4).

The CDC was ostensibly attempting to stymy future research efforts into Morgellons Disease and use their study as the end all of Morgellons research, but why? Clearly, one of the hallmarks of empirical research is the ability to replicate one’s findings over time.

Morgellons has historically been linked to chemtrails, genetically modified organisms (GMOs; a specific bacteria known as Agrobacterium)(4), Lyme Disease (1, 6, 9), immune weakness (1, 2) and environmental toxicity (1, 2, 6). According to researchers at the State University of New York, “Agrobacterium represents a universal gene and protein transfer machine” (7) and hence laboratory (genetically modified) creations now have the ability to alter the DNA of humans (4). Hence, agrobacterium allows horizontal transfer of DNA and would be a likely culprit in the creation of Morgellons although other factors appear to be involved.

Following up from the CDC study in January of 2012, new independent research just published in the Journal of Clinical and Experimental Dermatology Research found that Morgellons is in fact a real disease (8). Based on extensive physical, microscopic and fluorescence examination of skin, hair, tissues, calluses, etc of three Morgellons sufferers, the researchers reported several interesting findings:

  • Abnormal functioning of the follicular keratinocytes (genetic alterations in DNA caused hair follicles and skin to malfunction)
  • Fibers, which under the microscope (100x), had a unique floral arrangement and colors of blue, red, white, green and some brightly fluorescent
  • “Filaments stemming from the bulb end were found in some follicles and these appeared as rootlike growths”
  • Many hairs were flattened (like tape) rather than circular which was similar to the fibers.
  • Filaments originated from a layer of epithelial cells and grew out of the body and could not have originated from textiles or fabric
  • For the first time demonstrated that Morgellons filaments or fibers contain keratin (structural protein which makes up the outer layer of the skin) and hence the fibers were created within the body.
  • Alterations in keratinocyte (skin) expression is most likely due to spirochetal infection from Lyme Disease.

The filaments from the Morgellons patients were confirmed to contain keratin via immunohistological staining with antibodies specific for human keratins. This means that fibers present in the subjects were found to be biological in origin and are produced by keratinocytes (proteins in the outer layer of skin). The findings are consistent with the 2012 CDC publication stating that over 80% of non‐biopsy material taken from patients had a protein composition.

The researchers concluded that the fibers “are clearly biological and human in nature and are not implanted textile fibers”. It may be probable to assume that since these fibers under the microscope contain floral and root-like structures that the origin may be via a cross contamination of DNA from plants and humans by way of GMOs although future research would need to confirm this theory.

This study opens the door for future research which is desperately needed into the environmental and epigenetic causes of Morgellons Disease with Lyme Disease and GMOs being the likely areas of interest.

This study was called “a must read” by the faculty of 1000 (F1000) which is a global community of over 10,000 experts who find the top 2% of the best new research articles in the field of biology and medicine.

NutraSilver Ends Morgellons Suffering

Lucy  2009

“About six months ago, I did some research on the internet and ran across a website called NutraSilver. It was for Morgellons but also said that it can be used for Fungal infections. I was hesitant on trying it because of the price. I was desperate so I placed an order.

I first tried it orally for two weeks and did not have luck. I then called the NutraSilver company and it was discussed that I might use it topically. I first applied it on the spots and after 4 days or so, it started going away. However, I had to end up covering my entire lower legs in NutraSilver® to keep the Fungus from relocating. It took about 2 full weeks to completely go away, BUT It is gone…And I am so Thankful. What a nightmare!!!! I posted a photo of what my legs looked like before.”

NutraSilver is a Fast and Effective Morgellons Disease Treatment

What our customers tell us is when taking NutraSilver®, they heal and they heal quickly. We are not medical professionals; we are distributors trying to help people. We appreciate the dozens and dozens of testimonials from grateful people who use NutraSilver to treat their Morgellons disease and who after a few weeks have gone back to their previous lives without their non-healing lesions, brain-fog or most of the other debilitating symptoms. We sincerely hope you will try NutraSilver and experience for yourself the amazing healing.

If you have Morgellons disease, you know this is not a skin disease. You know that Morgellons DISEASE is deep within your body and you know intuitively that the only way to treat Morgellons disease is from the inside. Pushing it out makes more sense than covering it up with skin creams and lotions, right? Get NutraSilver now to eliminate your Morgellons symptoms.

FDA’s Crackdown on Nutritional Suppliments Thwarted by Voter Outrage

The FDA’s NDI (new supplement) guidance threatens our access to supplements and indeed could destroy the industry, leaving drugs with a total monopoly.

After adamantly digging in its heals and refusing to reconsider, FDA has now at last agreed that the draft NDI guidance requires substantial revision. This follows a meeting between FDA Commissioner Margaret Hamburg and Mike Taylor, Deputy Commissioner of Foods, with Senators Harkin and Hatch, longtime champions of natural health.

According to our sources, the FDA stated in the meeting that the guidance would be revised to account for the numerous comments and concerns, and released in draft form once again, which will then be open for public comment. This is exactly what we asked for!

The FDA’s decision is a dramatic reversal of the position taken in an earlier meeting with the Senators’ representatives, in which they adamantly refused this very same request. While we may never know how or why the FDA changed their position, it is very likely that they were responding to our sustained public messages opposing the current NDI draft guidance.

ANH-USA has also been working closely with the Appropriations Committee that controls the FDA’s purse strings. We recently helped secure report language in the Appropriations Committee censuring the FDA for prematurely enforcing the NDI draft guidance while supposedly still in draft. The report goes on to further state: “The Committee urges FDA to withdraw the July 2011 NDI draft guidance and re-engage the dietary supplement community to develop a new guidance on what constitutes NDI.” This appears to be another reason the FDA is finally listening.

While the FDA has not yet agreed to a specific timeline for revising the guidance, nor indicated what exactly will be revised, they did commit to collaborating with the dietary supplement industry to create a list of agreed-upon “grandfathered” dietary ingredients (ie. supplements). A “grandfathered” dietary ingredient is an ingredient marketed before 1994 and therefore not considered an NDI (new). Grandfathered dietary ingredients are therefore not subject to the NDI guidance. Currently, there is no authoritative list of “grandfathered” dietary ingredients, leaving the legal status of many dietary supplements in limbo.

Although an authoritative list of “grandfathered” dietary ingredients could be useful to the supplement industry, we fear that the FDA will use this as an opportunity to knock out supplements that were indeed on the market before 1994 but may not meet the FDA’s idea of proof. This is precisely what happened to pyridoxamine, one of only three natural and bioavailable forms of vitamin B6. Although we know that pyridoxamine was on the market before 1994, the FDA determined that there was not enough of the right kind of evidence to conclusively prove so, and removed the ingredient from the market.

ANH-USA will participate in this process to try to ensure that the list of “grandfathered” dietary ingredients is inclusive in order to maintain your access to as many supplements as possible. We will also try to prevent the FDA from using the list as an excuse to delay indefinitely their promise to revise the draft guidance.

Your Health Freedoms Are at Risk

Write to your Senator or Congressman and let them know that you want to enjoy the freedom to choose when it comes to making informed health decisions.  Protect your health freedoms by screaming at the top of your lungs until the government finally understands that we are determined to maintain our health freedoms. www.nutrasilver.com

MRSA Infections: Even Vancomycin Isn’t Working Any Longer

MRSA Kills

TAKE-HOME POINTS

■ Vancomycin had been the drug of choice for the treatment of methicillin- resistant (MRSA) infections for many years; however, recently its efficacy has been compromised.

■ Decreasing vancomycin suscepti­bilities, increasing incidence of MRSA in­fections, and emergence of vancomycin-intermediate and vancomycin-resistant strains leading to clinical failures have made it necessary to reevaluate the options for the treatment of serious MRSA infections.

■ Consideration of alternative drugs for the treatment of MRSA infections is warranted when deciding upon the appropriate antimicrobial therapy.

Methicillin-resistant Staph­ylococcus aureus (MRSA) is a frequent cause of serious infections, such as pneumonia, bacteremia, endocarditis, and osteomyelitis. The incidence of MRSA infections has changed in the past 20 years, with 
an increase in hospital-acquired MRSA 
infections worldwide and the emergence of a growing number of community-acquired MRSA infections.1 For many decades, IV vancomycin has been the drug of choice for the treatment of these infections.2 However, the emergence of clinical isolates of MRSA with reduced vancomycin susceptibilities and reports of clinical failures with vancomycin, despite appropriate therapeutic doses, have created the need for considering alternative drugs.3

CHANGES IN MRSA 
SUSCEPTIBILITY

Although MRSA was first isolated in 1961, it did not become a worldwide problem until the 1970s. Its emergence as a frequent pathogen led to increased use of vancomycin. A glycopeptide with antistaphylococcal activity, vancomycin disrupts cell wall synthesis and arrests bacterial growth. Vancomycin rapidly became the mainstay for the treatment of beta-lactam-resistant gram-positive infections, especially those due to MRSA.2 By 2003, however, reports began to appear of clinical failures with vancomycin therapy of infections caused by susceptible MRSA strains.4 Since then, many similar reports have been published in which vancomycin-susceptible MRSA strains were identified and clinical failures resulted despite monitoring and maintenance of trough concentrations in a range previously thought to predict clinical success.5

Susceptibility of a microorganism to a specific antimicrobial drug is determined by the mean inhibitory concentration (MIC), the lowest concentration of the drug that inhibits the visible growth of the microorganism after an overnight in vitro incubation. The lower the MIC, the more susceptible the microorganism. Previously, MRSA strains were considered susceptible to vancomycin if their growth was inhibited at an MIC of 4 µg/mL or less. Susceptibility breakpoints for vancomycin were established by the Clinical and Laboratory Standards Institute (CLSI) more than 25 years ago. Improved methods of detection and a growing body of clinical data resulted in a decrease in the vancomycin susceptibility breakpoint to 2 µg/mL or less in 2006.6 More recently, reports have demonstrated clinical failures with therapeutic doses of vancomycin for bacteremias and pneumonias due to vancomycin-susceptible MRSA strains for which the MIC was 2 µg/mL or less. This disturbing trend has led many to suggest that the time has come to redefine the MIC cutoff point even lower.5

Initially, MRSA infections were almost exclusively hospital-acquired. MRSA now accounts for about 60% of clinical isolates of S aureus strains found in ICUs in the United States.7 The nosocomial connection to MRSA infections was clearly established, but in the past decade, community outbreaks, including bacteremias and skin and soft tissue infections, began to appear. Through DNA sequence analysis, the origin of most of these emerging community strains was traced to hospital or long-term care facilities. Over time, there has been an incremental increase in the observed vancomycin MICs of clinical isolates of susceptible MRSA. Referred to as MIC creep, this increase has been correlated with clinical failures in the treatment of MRSA infections.8

BACTERICIDAL VS INHIBITORY 
CONCENTRATIONS

Use of an antimicrobial agent with bactericidal activity has been a long-standing concept for treatment of serious MRSA infection, but not all strains of MRSA exhibit a bactericidal response when exposed to vancomycin. Minimum bactericidal concentration (MBC) is the lowest drug concentration that kills a microorganism. Vancomycin tolerance is defined as an MBC:MIC ratio greater than 32. Such tolerance has been reported in up to 15% of wild-type MRSA strains with vancomycin-susceptible MICs; however, the poor bactericidal activity of vancomycin in these strains poses a potential risk for clinical failure.3 Although the clinical significance of vancomycin tolerance is not fully understood, it has been associated with clinical failures observed in gram-positive infections of cancer patients.9

In addition to rising MICs and clinical failures with vancomycin-susceptible MRSA strains, diminished or even resistant strains have emerged in the past 15 years. Vancomycin-intermediate S aureus (VISA) was first observed in Japan in 1997. Originally set at 8 µg/mL, the vancomycin breakpoint MIC for VISA was lowered by the CLSI in 2006 to the current value of 4 to 8 µg/mL.6 Vancomycin tolerance has been observed in up to 74% of VISA strains.3 Vancomycin-resistant S aureus (VRSA) was identified in the United States in 2002. The CLSI lowered the vancomycin breakpoint MIC of VRSA from its original value of 32 µg/mL or greater to 16 µg/mL or greater.6 Vancomycin tolerance or frank resistance is seen in 100% of VRSA strains.3 Fortunately, the incidence of VRSA has remained low.10

The authors work in the Department of Infectious Diseases, Infection Control and Employee Health, University of Texas M.D. Anderson Cancer Center, Houston. Roy Borchardt is a physician assistant and the department editor, and Ken Rolstonis a professor of medicine. The authors have indicated no relationships to disclose relating to the content of this article.

Natural Alternative found that kills MRSA

 MRSA is killing more people than AIDS!  

It is rampant in hospitals, gyms and within the community.  Most antibiotics are no longer effective against this kind of Staph infection, so medical science is frantically searching for a non-toxic solution.  In FDA-certified independent in-vitro lab tests, NutraSilver killed 1.33 billion cells of MRSA in 60 seconds at the rate of 99.9999% with only one drop. If you took MRSA Staph bacterium the same amount of MRSA and placed it in a vat of antibiotics, nothing would happen!  MRSA does not care about Methicillin or Vancomyacin any longer!

Different Approach, Better Results

They are trying to turn off MRSA’s ability to reproduce with antibiotics; we have succeeded by switching of MRSA’s ability to breath! You can read the entire lab report here.

“I am a 38 year old female, single parent… I was terrified, hopeless & knew that I was inching closer to the grave, I prepared my Last Will & Testament and I put a couple Life Insurance policies on myself. I was very depressed from it all. I couldn’t help but be anything but with the thought of leaving my son behind.

“Today…I sleep through the night, I am up early in the morning, no naps..I stay on the go, I am swimming 100 laps 6 days a week on top of my daily cardio & strength training. I haven’t had ANY energy to exercise since 2005!! I’m eating as a vegetarian now and since I ordered your product the first time around, I’ve lost 65 lbs that I had gained while I was down ill…with 20 more to go! 19 Hospital visit and NutraSilver was the only thing that helped eliminate MRSA from my body.

“NutraSilver has brought me back to LIFE. It’s literally wiped out ALL of my MRSA symptoms & I now keep it combined with a Pro-biotic and Golden Seal. I maintain taking NutraSilver at a dose 40 drops a day. My son and I cannot EVER thank you enough..YOU helped save my life when 19 hospital stays & the strongest of medication couldn’t even make a dent!

“YOU GUYS ARE ANGELS!!! THANK YOU!!! THANK YOU!!!”

Don’t Allow Ticks To Steal Your Life!!!

What ticks cause infections?

There are many different types of ticks in the United States, some of which are capable of transmitting infections. The risk of developing these infections depends upon the geographic location, season of the year, type of tick, and, for Lyme disease, how long the tick was attached to the skin.

While many people are concerned after being bitten by a tick, the risk of acquiring a tick-borne infection is quite low, even if the tick has been attached, fed, and is actually carrying an infectious agent. Ticks transmit infection only after they have attached and then taken a blood meal from their new host [1]. A tick that has not attached (and therefore has not yet become engorged from its blood meal) has not passed any infection. Since the deer tick that transmits Lyme disease must feed for >36 hours before transmission of the spirochete, the risk of acquiring Lyme disease from an observed tick bite, for example, is only 1.2 to 1.4 percent, even in an area where the disease is common.

If a person is bitten by a deer tick (the type of tick that carries Lyme disease), a healthcare provider will likely advise one of two approaches:

  • Observe and treat if signs or symptoms of infection develop
  • Treat with a preventive antibiotic immediately

There is no benefit of blood testing for Lyme disease at the time of the tick bite; even people who become infected will not have a positive blood test until approximately two to six weeks after the infection develops (post-tick bite).

The history of the tick bite will largely determine which of these options is chosen. Before seeking medical attention, the affected person or household member should carefully remove the tick and make note of its appearance. Only the Ixodes species of tick, also known as the deer tick, causes Lyme disease.

HOW TO REMOVE A TICK

The proper way to remove a tick is to use a set of fine tweezers and grip the tick as close to the skin as is possible. Do not use a smoldering match or cigarette, nail polish, petroleum jelly (eg, Vaseline), liquid soap, or kerosene because they may irritate the tick and cause it to behave like a syringe, injecting bodily fluids into the wound.

The proper technique for tick removal includes the following:

  • Use fine tweezers to grasp the tick as close to the skin surface as possible.
  • Pull backwards gently but firmly, using an even, steady pressure. Do not jerk or twist.
  • Do not squeeze, crush, or puncture the body of the tick, since its bodily fluids may contain infection-causing organisms.
  • After removing the tick, wash the skin and hands thoroughly with soap and water.
  • If any mouth parts of the tick remain in the skin, these should be left alone; they will be expelled on their own. Attempts to remove these parts may result in significant skin trauma.

AFTER THE TICK IS REMOVED

Tick characteristics — It is helpful if the person can provide information about the size of the tick, whether it was actually attached to the skin, if it was engorged (that is, full of blood), and how long it was attached.

  • The size and color of the tick help to determine what kind of tick it was
  • Ticks that are brown and approximately the size of a poppy seed or pencil point are deer ticks. These can transmit Borrelia burgdorferi (the bacterium that causes Lyme disease) and a number of other tick-borne infections, including babesiosis and anaplasmosis. Deer ticks live primarily in the northeast and mid-Atlantic region (Maine to Virginia) and in the midwest (Minnesota and Wisconsin) region of the United States, and less commonly in the western US (northern California).
  • Ticks that are brown with a white collar and about the size of a pencil eraser are more likely to be dog ticks (Dermacentor species). These ticks do not carry Lyme disease, but can rarely carry another tick-borne infection called Rocky Mountain spotted fever that can be serious or even fatal.
  • A brown to black tick with a white splotch on its back is likely a female Amblyomma americanum (Lone Star tick; named after the white splotch). This species of tick has been reported to spread an illness called STARI (southern tick-associated rash illness). STARI causes a rash that is similar to the erythema migrans rash, but without the other features of Lyme disease. Although this rash is thought to be caused by an infection, a cause for the infection has not yet been identified. This type of tick can also carry and transmit another infection called human monocytic ehrlichiosis.
  • A tick that was not attached, was easy to remove or just walking on the skin, and was still flat and tiny and not full of blood when it was removed could not have transmitted Lyme disease or any other infection since it had not yet taken a blood meal.
  • Only ticks that are attached and have finished feeding or are near the end of their meal can transmit Lyme disease. After arriving on the skin, the tick that spreads Lyme disease usually takes 24 hours before feeding begins.
  • Even if a tick is attached, it must have taken a blood meal to transmit Lyme disease. At least 36 to 48 hours of feeding is required for a tick to have fed and then transmit the bacterium that causes Lyme disease. After this amount of time, the tick will be engorged (full of blood). An engorged tick has a globular shape and is larger than an un-engorged one.

The organism that causes Lyme disease, B. burgdorferi, lies dormant in the inner aspect of the tick’s midgut. The organism becomes active only after exposure to the warm blood meal entering the tick’s gut. Once active, the organism enters the tick’s salivary glands. As the tick feeds, it must get rid of excess water through the salivary glands. Thus, the tick will literally salivate organisms into the wound, thereby passing the infection to the host.

Need for treatment — The clinician will review the description of the tick, along with any physical symptoms, to decide upon a course of action. The Infectious Diseases Society of America (IDSA) recommends preventive treatment with antibiotics only in people who meet ALL of the following criteria:

  • Attached tick identified as an adult or nymphal I. scapularis (deer) tick
  • Tick is estimated to have been attached for ≥36 hours (based upon how engorged the tick appears or the amount of time since outdoor exposure)
  • Antibiotic treatment can begin within 72 hours of tick removal
  • The local rate of tick infection with B. burgdorferi is ≥20 percent (known to occur in parts of New England, parts of the mid-Atlantic states, and parts of Minnesota and Wisconsin)
  • The person can take doxycycline (eg, the person is not pregnant or breastfeeding or a child <8 years of age)

If the person meets ALL of the above criteria, the recommended dose of doxycycline is a single dose of 200 mg for adults and 4 mg/kg, up to a maximum dose of 200 mg, in children ≥ 8 years.

If the person cannot take doxycycline, the IDSA does not recommend preventive treatment with an alternate antibiotic for several reasons: there are no data to support a short course of another antibiotic, a longer course of antibiotics may have side effects, antibiotic treatment is highly effective if Lyme disease were to develop, and the risk of developing a serious complication of Lyme disease after a recognized bite is extremely low.

When you no longer wish to take antibiotics…

You are here to find a solution to your current Lyme Disease infection. You have found it. NutraSilver simply works and we guarantee that for 60 days. Do not suffer another day. There is no risk to you because your results are guaranteed. Lyme disease done.

  • NutraSilver® is a fast and effective Lyme Disease Treatment

MONITORING FOR LYME DISEASE

Many people have incorrect information about Lyme disease. For example, some people are concerned that Lyme disease is untreatable if antibiotics are not given early (this is untrue; even later features of Lyme disease can be effectively treated with appropriate antibiotics). Many local Lyme disease networks and national organizations disseminate unproven information and should not be the sole source of education about Lyme disease.

Signs of Lyme disease — Whether or not a clinician is consulted after a tick bite, the person who was bitten (or the parents, if a child was bitten) should observe the area of the bite for expanding redness, which would suggest erythema migrans (EM), the characteristic rash of Lyme disease.

The EM rash is usually a salmon color although, rarely, it can be an intense red, sometimes resembling a skin infection. The color may be almost uniform. The lesion typically expands over a few days or weeks and can reach over 20 cm (8 inches) in diameter. As the rash expands, it can become clear (skin-colored) in the center. The center of the rash can then appear a lighter color than its edges or the rash can develop into a series of concentric rings giving it a “bull’s eye” appearance. The rash usually causes no symptoms, although burning or itching has been reported.

In people with early localized Lyme disease, EM occurs within one month of the tick bite, typically within a week of the tick bite, although only one-third of people recall the tick bite that gave them Lyme disease. Components of tick saliva can cause a short-lived (24 to 48 hours) rash that should not be confused with EM. This reaction usually does not expand to a size larger than a dime.

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